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Vaidya was supported by the National Institutes of Diabetes and Digestive and Kidney Disease of the National Institutes of Health under award R01 DK107407, by the National Heart, Lung, and Blood Institute under award K23 HL111771, and by grant 2015085 from the Doris Duke Charitable Foundation.

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This work was supported by grants from National Health Research Institutes (MG-093-PP-08 and MG-093-PP-07 to SF Huang and Y-R Chen, respectively), Department of Health (DOH94-TD-G-111-016 to Y-R Chen), and National Science Council, Taiwan (NHRI92A1-NSCCA16-5 to S-F Tsai).

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The exponential functions for K out in each tumor group were quite similar (see ), and thus, whatever the underlying mechanism, it is not unique to a particular tumor type.

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To identify specific genes or pathways that may directly or indirectly contribute to the protective effect of gefitinib on tumorigenesis, three to four independent tumors and normal tissues from gefitinib treatment and control groups were used for this analysis.

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NSCLC reported that EGFR activating mutations were the most important independent predictors for time to treatment failure compared with other mutations, among which exon 19 deletion and L858R mutations were the best predictors for longer time to treatment failure in a multivariate analysis ().

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In this study, the gene expression of MDR proteins (MRP1, MRP2, and MRP3) was induced by conventional chemotherapeutic agents in pancreatic carcinoma cell line, especially by gemcitabine in a dose-dependent manner.

Take L858R as example, which is a frequent EGFR mutation in NSCLC where leucine (L) is substituted by arginine (R) at the locus.

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To establish evidence-based recommendations for the molecular analysis of lung cancers that are that are required to guide EGFR- and ALK-directed therapies, addressing which patients and samples should be tested, and when and how testing should be performed.

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Tumor samples from patients who consented to have biomarkers assessed were analyzed at two central laboratories to determine biomarker status, with EGFR mutation status the first priority.

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If a person's tumor is negative for the most common KRAS mutation, tests for other less common mutations not detected by the current test may be used to help predict therapeutic responses.

OBJECTIVES: Epidermal growth factor receptor (EGFR) mutation-positive non-small cell lung cancer (NSCLC) is a specific lung cancer subtype characterized by sensitivity to treatment with EGFR tyrosine kinase inhibitors (TKIs).

Radiation myelopathy: estimates of risk in 1048 patients in three randomized trials of palliative radiotherapy for non-small cell lung cancer: the Medical Research Council Lung Cancer Working Party.

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